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Detailled Reference
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Article (Scientific journals)
High-Risk Clonal Hematopoiesis as the Origin of AITL and NPM1-Mutated AML.
Tiacci, Enrico
;
Venanzi, Alessandra
;
Ascani, Stefano
et al.
2018
•
In
The New England journal of medicine, 379
(10), p. 981-984
Peer reviewed
Permalink
https://hdl.handle.net/10993/53226
DOI
10.1056/NEJMc1806413
PubMed
30184450
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Keywords :
Hematopoiesis/genetics; Humans; Immunoblastic Lymphadenopathy/genetics; Leukemia, Myeloid, Acute/genetics/pathology; Lymphoma, T-Cell/genetics/pathology; Male; Middle Aged; Mutation; Neoplasms, Second Primary/genetics; Nuclear Proteins/genetics; Nucleophosmin
Disciplines :
Hematology
Author, co-author :
Tiacci, Enrico
Venanzi, Alessandra
Ascani, Stefano
Marra, Andrea
Cardinali, Valeria
Martino, Giovanni
Codoni, Veronica
;
Universita degli studi di Perugia > Institute of Hematology and Center for Hemato-Oncology Research
Schiavoni, Gianluca
Martelli, Maria Paola
Falini, Brunangelo
External co-authors :
yes
Language :
English
Title :
High-Risk Clonal Hematopoiesis as the Origin of AITL and NPM1-Mutated AML.
Publication date :
2018
Journal title :
The New England journal of medicine
ISSN :
0028-4793
eISSN :
1533-4406
Volume :
379
Issue :
10
Pages :
981-984
Peer reviewed :
Peer reviewed
Available on ORBilu :
since 19 December 2022
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