Circulating Glucocorticoids Shape Phasic and Tonic GABA Inhibition in Lamina II of Rat Spinal Cord.

Reference : Circulating Glucocorticoids Shape Phasic and Tonic GABA Inhibition in Lamina II of Ra...


	Document type :	Scientific congresses, symposiums and conference proceedings : Paper published in a book
	Discipline(s) :	Life sciences : Anatomy (cytology, histology, embryology...) & physiology
	To cite this reference:	http://hdl.handle.net/10993/14085

Title :	Circulating Glucocorticoids Shape Phasic and Tonic GABA Inhibition in Lamina II of Rat Spinal Cord.
Language :	English
Author, co-author :	Zell, Vivien [University of Luxembourg > Faculty of Language and Literature, Humanities, Arts and Education (FLSHASE) > Integrative Research Unit: Social and Individual Development (INSIDE) >]
	Anton, Fernand [University of Luxembourg > Faculty of Language and Literature, Humanities, Arts and Education (FLSHASE) > Integrative Research Unit: Social and Individual Development (INSIDE) >]
	Hanesch, Ulrike [University of Luxembourg > Faculty of Language and Literature, Humanities, Arts and Education (FLSHASE) > Integrative Research Unit: Social and Individual Development (INSIDE) >]
	Poisbeau, Pierrick [Institut des Neurosciences Cellulaires et Intégratives, Centre National de la Recherche Scientifique and University of Strasbourg, Strasbourg, France > Nociception and Pain Department]
	Darbon, Pascal [Institut des Neurosciences Cellulaires et Intégratives, Centre National de la Recherche Scientifique and University of Strasbourg, Strasbourg, France > Nociception and Pain Department]
Publication date :	2012
Main document title :	FENS Abstr., vol. 6, p153, 6.22, 2012
Peer reviewed :	Yes
Event name :	8th FENS Forum of Neuroscience
Event date :	14-07-2012 to 18-07-2012
Permalink :	http://hdl.handle.net/10993/14085

File(s) associated to this reference

Fulltext file(s):

	File	Commentary	Version	Size	Access
Limited access	FENS-barcelone-2012_Zell et al.pdf		Author preprint	90.42 kB	Request a copy

All documents in ORBi^lu are protected by a user license.