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See detailInvited talk - Modelle der dreimodalen Faktorenanalyse
Krolak-Schwerdt, Sabine UL

Scientific Conference (1991)

Detailed reference viewed: 16 (0 UL)
See detailInvited talk - Prädiktoren und prognostische Validität der Sekundarschulempfehlungen in Luxemburg
Krolak-Schwerdt, Sabine UL; Klapproth, Florian

Conference given outside the academic context (2012)

Detailed reference viewed: 19 (0 UL)
See detailInvited talk - Schulübergangsentscheidung in Luxemburg
Krolak-Schwerdt, Sabine UL

Conference given outside the academic context (2013)

Detailed reference viewed: 14 (0 UL)
See detailInvited talk - TRANSINTER - erste Ergebnisse und zukünftige Pläne
Pit-Ten Cate, Ineke UL; Glock, Sabine; Krolak-Schwerdt, Sabine UL

Conference given outside the academic context (2013)

Detailed reference viewed: 23 (1 UL)
See detailInvited talk - Übergangsentscheidungen in Luxemburg
Krolak-Schwerdt, Sabine UL; Pit-Ten Cate, Ineke UL; Glock, Sabine et al

Conference given outside the academic context (2016)

Detailed reference viewed: 31 (3 UL)
See detailInvited talk -Verarbeitung von personenbeschreibenden Texten
Krolak-Schwerdt, Sabine UL

Scientific Conference (2002)

Detailed reference viewed: 14 (0 UL)
Peer Reviewed
See detailDe invloed van John Dewey op de Nederlandse pedagogiek.
Biesta, Gert UL; Miedema, S.

in Pedagogische Verhandelingen. (1987), 10(2), 324-335

Detailed reference viewed: 268 (0 UL)
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Peer Reviewed
See detailInvoluntary eyelid closure after STN-DBS: evidence for different pathophysiological entities.
Weiss, Daniel; Wachter, Tobias; Breit, Sorin et al

in Journal of neurology, neurosurgery, and psychiatry (2010), 81(9), 1002-7

OBJECTIVE: Involuntary eyelid closure (IEC) may occur after deep brain stimulation of the subthalamic nucleus (STN-DBS) in Parkinson's disease (PD) and is often categorised as apraxia of lid opening (ALO ... [more ▼]

OBJECTIVE: Involuntary eyelid closure (IEC) may occur after deep brain stimulation of the subthalamic nucleus (STN-DBS) in Parkinson's disease (PD) and is often categorised as apraxia of lid opening (ALO), albeit the appropriateness of this term is under debate. To gain insight into the hitherto undefined pathophysiology of IEC after STN-DBS, we performed a comprehensive clinical and electrophysiological characterisation of lid function in a total of six PD patients. METHODS: The study was carried out in six PD patients who developed IEC after STN-DBS. They underwent neurological examination and electromyography recording of activity in the orbicularis oculi muscle (OO) upon varying stimulation patterns. Intraoperative studies were performed in one patient. RESULTS: Increasing STN-DBS intensity induced IEC in four patients, whereas it improved the condition in two. Needle EMG showed tonic hyperactivity of the OO in STN-DBS induced IEC, while variable patterns of OO activity (irregular and tonic) were seen in patients with STN-DBS-relieved IEC. Intraoperative analysis in one patient showed evidence for IEC being induced by activation of corticobulbar fibres. CONCLUSIONS: We identified two groups of IEC after STN-DBS based on clinical and EMG patterns: (1) STN-DBS induced IEC associated with tonic OO overactivity and (2) STN-DBS relieved IEC presenting with variable EMG patterns. Our findings provide relevant information on pathophysiology of STN-DBS related IEC and implications for its therapeutic management. [less ▲]

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See detailInvolvement and Community participation
Limbach-Reich, Arthur UL

Scientific Conference (2017, September 22)

Detailed reference viewed: 40 (2 UL)
Peer Reviewed
See detailInvolvement of alpha-synuclein in Parkinson's disease and other neurodegenerative disorders.
Krüger, Rejko UL; Muller, T.; Riess, O.

in Journal of neural transmission (Vienna, Austria : 1996) (2000), 107(1), 31-40

A major step in the elucidation of the pathogenesis of neurodegenerative disorders was the identification of a mutation in the alpha-synuclein gene in autosomal dominant Parkinson's disease (PD). Alpha ... [more ▼]

A major step in the elucidation of the pathogenesis of neurodegenerative disorders was the identification of a mutation in the alpha-synuclein gene in autosomal dominant Parkinson's disease (PD). Alpha-synuclein is the main component of Lewy bodies (LB), the neuropathological hallmark of PD. Moreover, a fragment of alpha-synuclein (NAC) is the second major component of amyloid plaques in Alzheimer's disease (AD). Recent studies of other neurodegenerative disorders such as dementia with LB (DLB), multiple system atrophy (MSA) and amyotrophic lateral sclerosis (ALS) also revealed intracellular accumulations of alpha-synuclein in affected brain regions. This may indicate that these disorders partially share common pathogenic mechanisms. Recent data provide first insights into the physiological function of alpha-synuclein and support the concept of an essential role of alpha-synuclein in neurodegeneration. Increasing knowledge on the pathogenic molecular mechanisms of neurodegeneration and of the pathophysiological function of alpha-synuclein in particular may influence future development of therapeutic strategies in neurodegenerative disorders. [less ▲]

Detailed reference viewed: 61 (0 UL)
Peer Reviewed
See detailInvolvement of catecholamine systems in the central effects of histamine on blood pressure
Philippu, Athineos; Hanesch, Ulrike UL; Dietl, Hans et al

in Uvnäs, B; Tasaka, T (Eds.) Advances in histamine research (1982)

Detailed reference viewed: 23 (0 UL)
Peer Reviewed
See detailInvolvement of nitric oxide in the sensitization of articular afferents to mechanical stimuli
Pawlak, Matthias; Meßlinger; Hanesch, Ulrike UL et al

in Regulatory Peptides (2000), 89

Detailed reference viewed: 18 (0 UL)
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See detailInvolvement of the cerebellum in Parkinson disease and dementia with Lewy bodies.
Seidel, Kay; Bouzrou, Mohamed; Heidemann, Nina et al

in Annals of neurology (2017), 81(6), 898-903

Brains from patients with Parkinson disease or dementia with Lewy bodies show aggregation of alpha-synuclein in precerebellar brainstem structures. Furthermore, patients exhibit resting tremor, unstable ... [more ▼]

Brains from patients with Parkinson disease or dementia with Lewy bodies show aggregation of alpha-synuclein in precerebellar brainstem structures. Furthermore, patients exhibit resting tremor, unstable gait, and impaired balance, which may be associated with cerebellar dysfunction. Therefore, we screened the cerebella of 12 patients with alpha-synucleinopathies for neuropathological changes. Cerebellar nuclei and neighboring white matter displayed numerous aggregates, whereas lobules were mildly affected. Cerebellar aggregation pathology may suggest a prionlike spread originating from affected precerebellar structures, and the high homogeneity between patients with dementia with Lewy bodies and Parkinson disease shows that both diseases likely belong to the same neuropathological spectrum. Ann Neurol 2017;81:898-903. [less ▲]

Detailed reference viewed: 122 (1 UL)